Knee Dislocation

If you remember one thing from this post remember this: up to 50% of dislocations spontaneously reduce before presentation to the the ER - these patients are STILL at high risk for neuromuscular injury. Take a good history about the mechanism of injury, get a good exam, and follow the instructions below, to make sure you dont miss a popliteal artery or peroneal nerve injury!


Diagnosis of Knee dislocation:

  • up to 50% of dislocations spontaneously reduce before presentation to the ER, but that doesn’t mean a neurovascular injury didn’t occur during the dislocation

    • consider the mechanism of injury: motor vehicle accidents, other high velocity mechanisms (falls, downhill skiing, football) make dislocation more likely

    • in rare cases, low velocity injuries in the obese, or sudden twisting motions in athletes can also result in dislocation

  • knee exam should focus on appearance, integrity/stability of joint, distal perfusion, and evaluating for peroneal nerve injury

    • peroneal nerve provides ankle dorsiflexion, toe extension, and sensation to first dorsal web space

    • usually 3 or more major knee ligaments must rupture for the knee to dislocation, so any knee exam w/multi-planar instability should be a suspected dislocation that spontaneously reduced

  • Anterior dislocation is most common (50-60%) named for the direction of translation of the proximal tibia

  • Posterior dislocation is even more commonly associated w/popliteal artery injury

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Diagnosis of complications, especially in a spontaneously reduced knee:

  • most common injury is popliteal artery injury

    • presence of pulses does not exclude injury to the popliteal artery

    • if missed, can end up with AKA (delay of pop artery repair beyond 8 hrs invariably leads to limb amputation)

    • this is a highly litigated injury

  • ruling a popliteal injury via “hard signs” requires an immediate vascular surgery consult

    • hard signs include absence of pulse, pale or dusky leg, parasthesias and paralysis, rapidly expanding hematoma, pulsatile bleeding, bruit or thrill over the wound

  • There is no physical exam sensitive enough to rule out popliteal injury!!

    • quality of evidence behind ABI is poor as well

    • Wills et al prospective study suggests that normal ABI + period of observation w/no change in exam is 100% sensitive combination for ruling out vascular injury

    • Standard angiography is the standard of care

      • CT angio with runoff is next best test in the ER - ORDER THIS if concern for vascular injury


Management of a currently dislocated knee:

1. First and foremost the immediate reduction, and if neurovascular compromise exists – without radiographs.

a. Look for an anteriomedial skin furrow or “pucker sign” when the knee is extended – this signifies a posteriolateral dislocation, which are not reducible by closed reduction (require open reduction in OR).

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a. Document neurovascular exam before and after reduction attempts

b. Initial approach should be application of longitudinal traction to lower leg (anterior dislocation may require additional lifting of distal femure, while posterior may require lifting the proximal tibia to complete reduction.

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c. After reduction, the knee should be immobilized in a long leg posterior splint with the knee in 15-20 degrees of flexion

d. Again, these pts need either normal ABI with close monitoring and serial exams OR CTA to rule out vascular injury after initial reduction

References:

http://www.emdocs.net/knee-dislocation-pearls-and-pitfalls/

https://www.emrap.org/episode/emrap20203/medicallegal

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TIA framework

I often have trouble wrapping my head around the concept of TIA. Thinking about it on a continuum with stroke, like the "unstable angina of the brain", caused by the same disease processes and modifiable risk factors as ischemic strokes, has helped me a lot. I have organized the main points into this graphic. I hope it helps you too.

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1. TIA exists on a continuum with stroke:

-Even symptoms that resolve after 24 hrs may have abnormality on diffusion-weighted MRI (much more advanced imaging than we had when 24 hr cutoff was developed)

-think of it like “unstable angina of the brain”

-15% of pt’s with TIA go on to have a larger stroke in next 3 months (and HALF of those happen in the first 48 hrs – so start treatment right away!)

-Treatment is same for TIA as for small strokes – namely, to prevent major stroke in future

 

2. Alternative nomenclature:

  • Stroke

    • Persistent deficits

    • Infarction on imaging

  • Transient Symptoms with Infarction (TSI)

    • Deficits resolve

    • Infarction (stroke) on imaging

  • Transient Ischemic Attack (TIA)

    • Deficits resolve

    • No Infarction (stroke) on imaging

3. Three key elements of TIA history:

-symptoms onset is SUDDEN

-symptom is a DEFICIT (or loss of something – such as sensation or strength)

– the presence of a paresthesia or abnormal smell etc. is more likely intracranial or migraine or seizure

-deficit is FOCAL and makes sense anatomically (correlates to a specific area of the brain or spinal cord – that’s right, the spinal cord can infarct too!)

            - multiple/generalized symptoms is usually something else.

 

4. Exam:

-always perform a complete neuro exam beyond just NIH scale

-thorough cardiac exam (afib? Pulse defecits?)

 

5. Unique stroke etiologies (other than most common atherosclerotic thromboembolic disease):

-Endocarditis > give IV Abx

-Carotid artery dissection > give AC

-Aortic arch dissection > CT surgery

-Temporal arteritis > corticosteroids

 

6. TIA mimics:

-focal/partial seizure

-todd’s paralysis

-complex migraine

-recrudescence triggered by other acute illness

-cardiac syncope

-metabolic (glucose, sodium, calcium)

-brain lesion

-demyelinating disease

-acute vestibular syndrome

-peripheral nerve lesion/neuropathy

-CNS infection

-psychogenic      

 

7. Initial evaluation/workup:

-Cardiac monitor (pick up Afib)

-EKG

-Labs (CBC, coags, often a troponin is warranted)

-CT head

-CTA head & neck for large vessel territory

-MRI (does not need to happen this second, but should be urgent, is often needed to confirm the diagnosis, which serves to encourage treatment (e.g. aspirin, statin, smoking cessation) so get it in the ER if you can)

 

8. Initial treatment in ER:

-162mg aspirin if no bleed on initial CT

                             -clopidogrel if ASA allergy

-neurology may recommend other anti-platelets (e.g. both ASA & plavix) but you dont have to start both yourself

-markedly elevated BP can be very slowly lowered (like really slowly. Like over days. PO meds not IV). Permissive hypertension is okay acutely because you want to make sure that ischemic brain gets perfused!

 

9. Who gets admitted?

-AHA says those who have symptoms <72 hrs ago, ABCD2 score >3, and those who are unable to get rapid evaluation as an outpt should be admitted

-Unfortunately, ACEP says that there is not adequate validation of ABCD2 score to make it a reliable SOLO tool for disposition, and I agree, but I figured I would still include it for you guys because it can be used in concert with other risk factors/circumstances (such as co-morbidities, access to quick followup) to determine who should stay to have their MRI/carotid doppler/echo/cardiac monitoring ASAP vs. who can followup w/neuro and cards in the office. 

 

10. Who gets anticoagulation?

-Afib

-ventricular thrombi on echo

-CHADS-VAsc 1 or more in men, 2 or more in women

11. Other treatment (not in ER):

-assess and treat modifiable risk factors (HTN, HLD, DM, smoking)

-carotid stenosis imaging can indicate need for carotid endarterectomy which greatly reduces future TIA/stroke risk, this is probably the most important f/u test to get!

-echo can find Left side thrombus that requires long-term AC.  Can also find PFO. 



References:

https://www.emrap.org/episode/c3tia/c3tia

Blum CA, Kasner SE. Transient Ischemic Attacks Presenting with Dizziness or Vertigo. Neurol Clin 33 (2015) 629–642. PMID: 26231276

Coutts SB. Diagnosis and Management of Transient Ischemic Attack. Continuum (Minneap Minn) 2017;23(1):82–92.PMID: 28157745

Lo BM, Carpenter CR.  Clinical Policy: Critical Issues in the Evaluation of Adult Patients With Suspected Transient Ischemic Attack in the Emergency Department Ann Emerg Med. 2016;68:354-370.PMID: 27568419

Tarnutzer AA, Lee S, et al.  ED misdiagnosis of cerebrovascular events in the era of modern neuroimaging. Neurology 2017;88:1468–1477.PMID: 28356464

Easton JD, Saver JL, Albers GW, et al. Definition and evaluation of transient ischemic attack. Stroke. 2009 Jun 1;40(6):2276-93.PMID: 19423857

Prabhakaran S, Silver AJ, Warrior L, et al. Misdiagnosis of transient ischemic attacks in the emergency room. Cerebrovascular Diseases. 2008;26(6):630-5.PMID: 18984948

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EMS Protocol of the Week - Seizures

So, you’re having a seizure. What do you do? 

 

Well nothing, I guess; you’re having a seizure. 

 

But paramedics can do stuff! And with this brilliant and natural segue, we’re on to Protocol 513 – Seizures.

 

The ALS protocol for seizures does what it can to diagnose and treat within the confines of the paramedic’s scope of practice. They should be checking a 3-lead for any concerning arrhythmias that may be mimicking as seizure activity, as well as assessing for hypoglycemia that may need to be reversed. Notice that there is a note included that reminds providers to account for a relative hypoglycemia for diabetic patients who may be euglycemic by non-diabetic standards. Similar to other protocols where hypoglycemia is mentioned, ALS will respond by giving dextrose IV/IO or glucagon IM if unable to obtain vascular access.

 

From here, the protocol discusses benzos, offering separate IV/IO and IM/IN dosing strategies to account for the time and safety concerns that often come with attempting to secure vascular access in a patient that is actively thrashing about. Check the protocol for specifics, but broadly, ALS providers are allowed to administer up to two doses of lorazepam OR up to two doses of diazepam OR a single dose of midazolam by Standing Order. As an OLMC physician, you can authorize repeat doses of any of those as you see fit.

 

That’s it! Stop the seizure! Don’t forget that benzos will require a Tracking Number (MMC-####), but don’t be alarmed if the crew asks to call back for the number after they’ve controlled the seizure in front of them; they won’t leave you hanging! While you wait for their call back, you can brush up on the protocols at www.nycremsco.org or with the protocol binder!

Dave

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